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Pathways and gene networks mediating the regulatory effects of cannabidiol， a nonpsychoactive cannabinoid， in autoimmune T cells.

J Neuroinflammation. 2016;

Kozela Ewa，Juknat Ana，Gao Fuying，Kaushansky Nathali，Coppola Giovanni，Vogel

Products/Services Used	Details	Operation
Peptide Synthesis	Methods Reagents Lyophilized MOG35-55 peptide [MEVGWYRSPFSRVV HLYRNGK] purchased from GenScript (Piscataway, NJ, USA) was reconstituted in sterile PBS and the stock so- lution stored in aliquots at −20 °C.	Get A Quote

摘要

Our previous studies showed that the non-psychoactive cannabinoid， cannabidiol (CBD)， ameliorates the clinical symptoms in mouse myelin oligodendrocyte glycoprotein (MOG)35-55-induced experimental autoimmune encephalomyelitis model of multiple sclerosis (MS) as well as decreases the memory MOG35-55-specific T cell (TMOG) proliferation and cytokine secretion including IL-17， a key autoimmune factor. The mechanisms of these activities are currently poorly understood.