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Vitamin D attenuates human gingival fibroblast inflammatory cytokine production following advanced glycation end product interaction with receptors for AGE.

J. Periodont. Res.. 2018-10;

ElenkovaMartina，TiptonDavid A，KarydisAnastasios，SteinSidn

Products/Services Used	Details	Operation
Proteins, Expression, Isolation and Analysis	… Endotoxin levels in G‐HSA and HSA solutions (125 μg/mL) were determined using the ToxinSensor™ Endotoxin Detection System (GenScript; Piscataway, NJ). Solutions were prepared using endotoxin‐free water, vials, and tips provided in the kit …	Get A Quote

摘要

Vitamin D [1，25(OH) D or 1，25D3] is critical in musculoskeletal health， inflammation， immune response， and glucose metabolism. Patients with vitamin D deficiency may be at higher risk of diabetes and periodontitis. Diabetic patients exhibit exacerbated inflammation and more periodontal destruction. Advanced glycation end products (AGEs)， formed during diabetic hyperglycemia， activate inflammatory pathways in periodontitis. Human gingival fibroblasts (HGFs) express receptors for AGEs (RAGEs) and can contribute to inflammation.