Human cMyBP-C Antibody (2G2F3)[Biotin], mAb, Mouse
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cMyBP-C is not essential for sarcomere formation during embryogenesis, but is crucial for sarcomere organization and maintenance of normal cardiac function. Absence of cMyBP-C results in severe cardiac hypertrophy, increased heart-weight-to-body-weight-ratios, enlargement of ventricles, increased myofilament Ca²⁺ sensitivity and depressed diastolic and systolic function. GenScript Anti-Human cMyBP-C Antibody (2G2F3)[Biotin], mAb, Mouse is produced from the hybridoma resulting from fusion of SP2/0-Ag14 myeloma and B-lymphocytes obtained from a mouse immunized with recombinant human cMyBP-C expressed in mammalian cells.
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